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Compared to #BiomassBurning smoke-exposed women, smokers have poor #LungFunction & high IL-1β levels in #sputum. Women with biomass-burning smoke exposure present an increased abundance of Bacteroidota & Fusobacteriota. #microbiome #lung
onlinelibrary.wiley.com/shar…
Pneumogenomics Laboratory, INER retweeted
☕️ El café es maravilloso, pero también tiene algunos riesgos.
➡️Estudios han demostrado que el café puede aumentar el colesterol malo (LDL).
🚨Es mejor tomar café filtrado.
nmcd-journal.com/action/show…
Pneumogenomics Laboratory, INER retweeted
Chronic #biomass-burning smoke exposure is associated with decreased levels of #DNA #methylation at the #CpG cg23771366 site in PRSS23, reinforcing the relationship between PRSS23 and particulate matter. #COPD @HLA_Laboratory
mdpi.com/3246518 #mdpitoxics vía @@Toxics_MDPI – at Tlalpan, Distrito Federal
In this paper from our lab identified that chronic biomass-burning smoke exposure is associated with decreased levels of DNA methylation at the CpG cg23771366 site in PRSS23, between PRSS23.
mdpi.com/3246518 #mdpitoxics vía @@Toxics_MDPI – at Tlalpan, Distrito Federal
Pneumogenomics Laboratory, INER retweeted
A nice paper! In this study @HLA_Laboratory showed that SNPs in PADI2 and PADI4 have influence on PAD2, PAD4, and H3Cit levels, which are predictor markers of requirement IMV and non-survival in severe ARDS by SARS-CoV-2 pneumonia.
Read it!
doi.org/10.1016/j.cca.2025.1…
Pneumogenomics Laboratory, INER retweeted
Emerging links between phase separation and transcription factor haploinsufficiency dlvr.it/TFmJSs
Pneumogenomics Laboratory, INER retweeted
Viral infections exacerbate and accelerate the progression of chronic respiratory diseases, by disrupting immune responses and triggering inflammatory pathways. pubmed.ncbi.nlm.nih.gov/3945…
Pneumogenomics Laboratory, INER retweeted
Happy to have presented my poster at #ICLAF2024, highlighting the changes in treatment patterns between the EPIMAR 1 and 2 cohorts in autoimmune ILD. A great opportunity to share these findings with colleagues. Looking forward to further improving outcomes in autoimmune ILD
This research from our laboratory in collaboration with @INERsticio
Identified #MUC5B rs35705950 in #Antisynthetase Syndrome with #ILD associated with Usual Interstitial Pneumonia. mdpi.com/2999466 #mdpijcm vía @JCM_MDPI – at Tlalpan, Distrito Federal
Pneumogenomics Laboratory, INER retweeted
#Tongue #Tuberculosis as a Complication of Pott’s Disease in a Patient on Systemic #Steroid Therapy without Pulmonary Tuberculosis mdpi.com/2904754 #mdpimedicina a través de @MDPIOpenAccess – at Tlalpan, Distrito Federal
In this paper, our lab's Dr. @Gloria_PR23 identified the #TLR4 rs4986790 common allele and the A–C haplotype (rs4986790–rs4986791) as associated with a higher COPD risk in smokers; #COPD patients carrying the AA genotype showed increased pro-inflammatory cytokines. – at Tlalpan, Distrito Federal
New Research: Altered levels of IFN-γ, IL-4, and IL-5 depend on the TLR4 rs4986790 genotype in COPD smokers but not those exposed to biomass-burning smoke frontiersin.org/articles/10.… #FrontiersIn #Immunology
Pneumogenomics Laboratory, INER retweeted
Dr. Bautista-Becerril from @HLA_Laboratory led an interesting case report describing Tongue Tuberculosis as a Complication of Pott’s Disease in a Patient on Systemic Steroid Therapy without Pulmonary Tuberculosis mdpi.com/2904754 #mdpimedicina a través de @MDPIOpenAccess
#Tongue #Tuberculosis as a Complication of Pott’s Disease in a Patient on Systemic #Steroid Therapy without Pulmonary Tuberculosis mdpi.com/2904754 #mdpimedicina a través de @MDPIOpenAccess – at Tlalpan, Distrito Federal
Dr. @Gloria_PR23 from our Lab identified #TLR4 rs4986790 common allele, and the A–C #haplotype (rs4986790–rs4986791) are associated with a higher #COPD risk in smokers; COPD patients carrying the AA #genotype showed increased pro-inflammatory #cytokines
frontiersin.org/journals/imm…
Dr. Chávez-Galán @ChavezGalan78 is a guest co-editor of the Cell Biology of Infection in BMC Microbiology collection, which warrants high-quality papers! Submissions are open. – at Coyoacán, Distrito Federal
Dear colleagues, I am a guest editor of a special issue in BMC Microbiology.
Send us your works where you study the complexities of the pathogens/ cell host interactions.
#bmcmicrobiology #pathogens #immunology #flowcytometry
biomedcentral.com/collection…
Pneumogenomics Laboratory, INER retweeted
Los criterios de #Light han muerto. #RIP
DUETS ≥2 indicated high likelihood for exudate (PPV 98.8%, NPV 100%) with 1% misclassification versus 6.9% using Light’s criteria (p < 0.001).
onlinelibrary.wiley.com/doi/…
Pneumogenomics Laboratory, INER retweeted
The authors identified an association of HLA-DQB1*03:01 with predisposition to, and DQB1*03:02 with resistance to, bronchiectatic AD or #emphysema in RA.
Pneumogenomics Laboratory, INER retweeted
En ALAT Chile, se llevó a cabo el 1 foro de mujeres líderes en la medicina respiratoria. Coordinado por Patricia Schonffeldt que contó con la participación de 30 mujeres profesionales de LATAM
@ALATorax @ALATIntersticio @mariamolinao @IrinaPetracheMD @LydianaAvila @leridapadron
In our weekly seminar @Emmanuel_1311 presented the manuscript Association of #HLA alleles with chronic lung diseases in #rheumatoid #arthritis. academic.oup.com/rheumatolog… – at Tlalpan, Distrito Federal
The authors identified an association of HLA-DQB1*03:01 with predisposition to, and DQB1*03:02 with resistance to, bronchiectatic AD or #emphysema in RA.