Bell-Lab: Cardiac Disease Mechanisms retweeted
An insightful panel session featuring @LabBell @k8weeks @jameschong_doc @alta_schutte. The session unpacked the value of undertaking editorial & service roles in scientific journals and how EMCIs can leverage the skills acquired for future opportunities. #ishr_aus #csanz26
Bell-Lab: Cardiac Disease Mechanisms retweeted
“Together, we innovate.” 🧬
@escardio @EWGCCEChair is funding collaboration across borders
If you’re an #ECR in #cellular #cardiac #electrophysiology, this is your chance to build connections & push your science forward.
escardio.org/Research/Resear…
Deadline- 8 Dec 2025.
Bell-Lab: Cardiac Disease Mechanisms retweeted
🚨 Conference registration closes on 13th August! 🚨
Last chance to register for our meeting, 'Cross-Talk of Cells in the Heart 2025: Novel Mechanisms of Disease and Arrhythmias'.🫀
Programme and registration link here: physoc.org/events/cross-talk…
@danmjohnson @unibirmingham
Bell-Lab: Cardiac Disease Mechanisms retweeted
🌟 Join us on 3-4 Sept @unibirmingham for our meeting, ‘Cross-Talk of Cells in the Heart 2025’.
🫀 Discover novel mechanisms of disease and arrhythmias with leading experts.
Save now with early bird registration ⬇️ buff.ly/Z3Lvs0c
#HeartCrossTalk2025 #HeartResearch
Bell-Lab: Cardiac Disease Mechanisms retweeted
📢 Final call to present your research at our meeting, ‘Cross-Talk of Cells in the Heart 2025’. Submit your abstracts to showcase your findings on the mechanisms of heart disease & arrhythmias.
📝 Submit by 2 June ⬇️ buff.ly/jY1h99h
@MollyOReillyy @chrisRJO
Bell-Lab: Cardiac Disease Mechanisms retweeted
Upcoming Abstract Deadline!
Interested in attending the Cardiac Cellular Cross Talk meeting in Birmingham in September?
Submit your abstract before the 2nd June! Its going to be a good one!
physoc.org/events/cross-talk…
@MollyOReillyy @chrisRJO @OU_STEM @physoc @JPhysiol
Bell-Lab: Cardiac Disease Mechanisms retweeted
Johannes V. Janssens (@UniMelbMDHS) et al. found that mechanical loading reveals an intrinsic #cardiomyocyte stiffness contribution to diastolic dysfunction in murine cardiometabolic disease 🫀
📜 Read it here: buff.ly/3VGYF3G
Bell-Lab: Cardiac Disease Mechanisms retweeted
Bell-Lab: Cardiac Disease Mechanisms retweeted
Signing off from #CSANZ2024! What a great conference sharing the amazing research taking place across Australasia and from our international guests.
Already looking forward to the ISHR World congress in Nara 🇯🇵 2025
@ISHR_ECI
Bell-Lab: Cardiac Disease Mechanisms retweeted
In a new Clinical Outlook, @JonKalmanEP and @LMaizels discuss pulsed-field ablation, which is rapidly revolutionizing the field of ablation for atrial fibrillation: rdcu.be/dLW97
Really pleased to share our latest research published in @JMCCPlus. A collaborative project between Melbourne (@LabBell, @DelbridgeLea) and Birmingham (@PavlovicDavor), assessing the acute influence of sex steroids on atrial electrophysiology.
Looking for some weekend reading? 📜
Check out the latest paper from Wells et al with contributions from the @LabBell @DelbridgeLea @PavlovicDavor and @k8weeks
Is the electrophysiological response to sex steroids in the atria sex specific ?
shorturl.at/USjOe
Thank you to the Reviewers & Handling Editor @JMCCPlus – excellent input received in the development of the manuscript and a rapid/supportive process from submission through to publication.
Bell-Lab: Cardiac Disease Mechanisms retweeted
Thank you to @delbridgelea and @KimMellor_UAkl for our beautiful April cover image, of glycogen macromolecules captured by an enveloping autophagosome in the para-myofilament cell region.
Bell-Lab: Cardiac Disease Mechanisms retweeted
Review by Janssens et al. @UniMelbMDHS characterizes the concept of cardiomyocyte stiffness and develops an interpretation of “stiffness” attributes at the cellular and molecular levels. ow.ly/C8gf50QUkZN
#MyocardialStiffness
Bell-Lab: Cardiac Disease Mechanisms retweeted
Congratulations to Dr @marijajelinic and the team at @latrobe University for receiving a 2023 Vanguard Grant! Their project on understanding the cellular and molecular drivers of heart attacks is set to make significant contributions to cardiovascular research. 🌟
Pleased to share our most recent work. High-fat feeding abolishes transmural conduction gradients in ventricular slices. Epicardial conduction slowing strongly correlates with cardiomyocyte lipid accumulation. Novel arrhythmogenic mechanism in settings of cardiac adiposity?
Localized cardiomyocyte lipid accumulation is associated with slowed epicardial conduction in rats. A new Communication from Simon Wells, Lea Delbridge, James Bell @UniMelb @latrobe and colleagues: bit.ly/3t9F9S4
#CellularPhysiology
Work culminating from Simon Wells’ PhD, with great team effort from Hanneke Raaijmakers, Claire Curl, @chrisRJO, @scisarahh, @KimMellor_UAkl, @JonKalmanEP, Paulus Kirchhof, @PavlovicDavor, @CardiacPhenomic






























